A Pathway of Double-Strand Break Rejoining Dependent upon ATM, Artemis, and Proteins Locating to γ-H2AX Foci
Top Cited Papers
- 1 December 2004
- journal article
- research article
- Published by Elsevier BV in Molecular Cell
- Vol. 16 (5), 715-724
- https://doi.org/10.1016/j.molcel.2004.10.029
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Pathways of DNA Double-Strand Break Repair during the Mammalian Cell CycleMolecular and Cellular Biology, 2003
- V(D)J Recombination: RAG Proteins, Repair Factors, and RegulationAnnual Review of Biochemistry, 2002
- Genomic Instability in Mice Lacking Histone H2AXScience, 2002
- Hairpin Opening and Overhang Processing by an Artemis/DNA-Dependent Protein Kinase Complex in Nonhomologous End Joining and V(D)J RecombinationCell, 2002
- Artemis, a Novel DNA Double-Strand Break Repair/V(D)J Recombination Protein, Is Mutated in Human Severe Combined Immune DeficiencyCell, 2001
- Dose rate effectiveness and potentially lethal damage repair in normal and double-strand break repair deficient murine cells by γ-rays and 5-fluorouracilCancer Letters, 1998
- VDJ recombinationImmunology Today, 1992
- Cell-Cycle-Dependent Repair of Potentially Lethal Damage in the XR-1 γ-Ray-Sensitive Chinese Hamster Ovary CellRadiation Research, 1988
- Comparative Human Cellular Radiosensitivity: I. The Effect of SV40 Transformation and Immortalisation on the Gamma-irradiation Survival of Skin Derived Fibroblasts from Normal Individuals and from Ataxia-telangiectasia Patients and HeterozygotesInternational Journal of Radiation Biology, 1988
- The Repair of Potentially Lethal Damage in X-irradiated Cultures of Normal and Ataxia Telangiectasia Human FibroblastsInternational Journal of Radiation Biology and Related Studies in Physics, Chemistry and Medicine, 1981