Increased butyrate priming in the gut stalls microbiome associated-gastrointestinal inflammation and hepatic metabolic reprogramming in a mouse model of Gulf War Illness
- 1 July 2018
- journal article
- research article
- Published by Elsevier BV in Toxicology and Applied Pharmacology
- Vol. 350, 64-77
- https://doi.org/10.1016/j.taap.2018.05.006
Abstract
No abstract availableKeywords
Funding Information
- Gulf War Illness Research (W81XWH-13-2-0072)
- University of South Carolina School of Medicine
- University of Georgia College of Veterinary Medicine
- University of South Carolina
This publication has 68 references indexed in Scilit:
- Stearic Acid Accumulation in Macrophages Induces Toll-Like Receptor 4/2-Independent Inflammation Leading to Endoplasmic Reticulum Stress–Mediated ApoptosisArteriosclerosis, Thrombosis, and Vascular Biology, 2012
- Inflammasome-mediated dysbiosis regulates progression of NAFLD and obesityNature, 2012
- NF-κB controls energy homeostasis and metabolic adaptation by upregulating mitochondrial respirationNature, 2011
- Selected Health Conditions Among Overweight, Obese, and Non-Obese Veterans of the 1991 Gulf War: Results from a Survey Conducted in 2003-2005The Open Epidemiology Journal, 2011
- Curcumin prevents leptin raising glucose levels in hepatic stellate cells by blocking translocation of glucose transporter‐4 and increasing glucokinaseBritish Journal of Pharmacology, 2010
- Physical, Psychological, and Functional Comorbidities of Multisymptom Illness in Australian Male Veterans of the 1991 Gulf WarAmerican Journal of Epidemiology, 2009
- Nonalcoholic fatty liver disease: An overview of current insights in pathogenesis, diagnosis and treatmentWorld Journal of Gastroenterology, 2008
- Competitive inhibition of histone deacetylase activity by trichostatin A and butyrateBiochemistry and Cell Biology, 2007
- Activation of Toll-like receptor 4 is associated with insulin resistance in adipocytesBiochemical and Biophysical Research Communications, 2006
- Butyrate inhibits inflammatory responses through NFkappa B inhibition: implications for Crohn's diseaseGut, 2000