INCREASED SERUM AMYLOID A LEVELS REFLECT COLITIS SEVERITY AND PRECEDE AMYLOID FORMATION IN IL-2 KNOCKOUT MICE
- 30 September 2000
- journal article
- Published by Elsevier BV in Cytokine
- Vol. 12 (9), 1337-1347
- https://doi.org/10.1006/cyto.2000.0716
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- INTERLEUKIN-1 RECEPTOR ANTAGONIST: Role in BiologyAnnual Review of Immunology, 1998
- Enterocolitis and colon cancer in interleukin-10-deficient mice are associated with aberrant cytokine production and CD4(+) TH1-like responses.JCI Insight, 1996
- Anti-Amyloid DrugsDrugs & Aging, 1996
- Immunopathology of interleukin (IL) 2-deficient mice: thymus dependence and suppression by thymus-dependent cells with an intact IL-2 gene.The Journal of Experimental Medicine, 1995
- T cells, but not B cells, are required for bowel inflammation in interleukin 2-deficient mice.The Journal of Experimental Medicine, 1995
- Evidence that CD4+, but not CD8+ T cells are responsible for murine interleukin‐2‐deficient colitisEuropean Journal of Immunology, 1995
- Normal clonal expansion but impaired Fasmediated cell death and anergy induction in interleukin‐2‐deficient miceEuropean Journal of Immunology, 1995
- Direct Evidence for Circulating apoSAA as the Precursor of Tissue AA Amyloid DepositsScandinavian Journal of Immunology, 1988
- Amyloid A gene family expression in different mouse tissues.The Journal of Experimental Medicine, 1986
- Amyloidogenesis. One serum amyloid A isotype is selectively removed from the circulation.The Journal of Experimental Medicine, 1986