An N-Terminal Mutation of the Foxp3 Transcription Factor Alleviates Arthritis but Exacerbates Diabetes
- 25 May 2012
- journal article
- research article
- Published by Elsevier BV in Immunity
- Vol. 36 (5), 731-741
- https://doi.org/10.1016/j.immuni.2012.04.007
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Loss of Epigenetic Modification Driven by the Foxp3 Transcription Factor Leads to Regulatory T Cell InsufficiencyImmunity, 2012
- Control of TH17/Treg Balance by Hypoxia-Inducible Factor 1Cell, 2011
- Structure of a Domain-Swapped FOXP3 Dimer on DNA and Its Function in Regulatory T CellsImmunity, 2011
- An incoherent regulatory network architecture that orchestrates B cell diversification in response to antigen signalingMolecular Systems Biology, 2011
- Histone acetyltransferase mediated regulation of FOXP3 acetylation and Treg functionCurrent Opinion in Immunology, 2010
- The aryl hydrocarbon receptor interacts with c-Maf to promote the differentiation of type 1 regulatory T cells induced by IL-27Nature Immunology, 2010
- Gut-Residing Segmented Filamentous Bacteria Drive Autoimmune Arthritis via T Helper 17 CellsImmunity, 2010
- Regulatory T Cells Reinforce Intestinal HomeostasisImmunity, 2009
- The transcription factor T-bet controls regulatory T cell homeostasis and function during type 1 inflammationNature Immunology, 2009
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cellsNature, 2006