RBM5, 6, and 10 Differentially Regulate NUMB Alternative Splicing to Control Cancer Cell Proliferation
Top Cited Papers
- 11 December 2013
- journal article
- research article
- Published by Elsevier BV in Molecular Cell
- Vol. 52 (5), 720-733
- https://doi.org/10.1016/j.molcel.2013.11.010
Abstract
No abstract availableKeywords
This publication has 67 references indexed in Scilit:
- The RNA-binding landscapes of two SR proteins reveal unique functions and binding to diverse RNA classesGenome Biology, 2012
- Alternative pre-mRNA splicing regulation in cancer: pathways and programs unhingedGenes & Development, 2010
- Deciphering the splicing codeNature, 2010
- Aberrant alternative splicing and extracellular matrix gene expression in mouse models of myotonic dystrophyNature Structural & Molecular Biology, 2010
- Mechanisms of alternative splicing regulation: insights from molecular and genomics approachesNature Reviews Molecular Cell Biology, 2009
- RBM5/Luca-15/H37 Regulates Fas Alternative Splice Site Pairing after Exon DefinitionMolecular Cell, 2008
- Molecular analysis of deletions in human chromosome 3p21 and the role of resident cancer genes in diseaseBriefings in Functional Genomics and Proteomics, 2007
- Functional spliceosomal A complexes can be assembled in vitro in the absence of a penta-snRNPRNA, 2006
- Protein Composition and Electron Microscopy Structure of Affinity-Purified Human Spliceosomal B Complexes Isolated under Physiological ConditionsMolecular and Cellular Biology, 2006
- Chromosome 19 Translocation, Overexpression of Notch3, and Human Lung CancerJNCI Journal of the National Cancer Institute, 2000