Variation in the miRNA-433 Binding Site of FGF20 Confers Risk for Parkinson Disease by Overexpression of α-Synuclein
Open Access
- 8 February 2008
- journal article
- research article
- Published by Elsevier BV in American Journal of Human Genetics
- Vol. 82 (2), 283-289
- https://doi.org/10.1016/j.ajhg.2007.09.021
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Parkinson's disease: piecing together a genetic jigsawBrain, 2003
- Preferential neurotrophic activity of fibroblast growth factor‐20 for dopaminergic neurons through fibroblast growth factor receptor‐1cJournal of Neuroscience Research, 2003
- Regulation of α‐synuclein by bFGF in cultured ventral midbrain dopaminergic neuronsJournal of Neurochemistry, 2003
- Parkinson-Like Neurodegeneration Induced by Targeted Overexpression of α-Synuclein in the Nigrostriatal SystemJournal of Neuroscience, 2002
- Overexpression of human α-synuclein causes dopamine neuron death in primary human mesencephalic cultureBrain Research, 2002
- Complete Genomic Screen in Parkinson DiseaseJama-Journal Of The American Medical Association, 2001
- Muscarinic Receptor Stimulation Induces Translocation of an α-Synuclein Oligomer from Plasma Membrane to a Light Vesicle Fraction in CytoplasmPublished by Elsevier BV ,2001
- Correcting for a Potential Bias in the Pedigree Disequilibrium TestAmerican Journal of Human Genetics, 2001
- FGF-20, a Novel Neurotrophic Factor, Preferentially Expressed in the Substantia Nigra Pars Compacta of Rat BrainBiochemical and Biophysical Research Communications, 2000
- GOLD—Graphical Overview of Linkage DisequilibriumBioinformatics, 2000