Interaction of myogenic factors and the retinoblastoma protein mediates muscle cell commitment and differentiation
- 1 February 1993
- journal article
- research article
- Published by Elsevier BV in Cell
- Vol. 72 (3), 309-324
- https://doi.org/10.1016/0092-8674(93)90110-c
Abstract
No abstract availableKeywords
This publication has 78 references indexed in Scilit:
- Functional antagonism between c-Jun and MyoD proteins: A direct physical associationCell, 1992
- Cyclin A and the retinoblastoma gene product complex with a common transcription factorNature, 1991
- The E2F transcription factor is a cellular target for the RB proteinCell, 1991
- The retinoblastoma protein copurifies with E2F-I, an E1A-regulated inhibitor of the transcription factor E2FCell, 1991
- Aberrant regulation of MyoD1 contributes to the partially defective myogenic phenotype of BC3H1 cells [published erratum appears in J Cell Biol 1990 Jun;110(6):2231]The Journal of cell biology, 1990
- The protein Id: A negative regulator of helix-loop-helix DNA binding proteinsCell, 1990
- Suppression of Tumorigenicity of Human Prostate Carcinoma Cells by Replacing a Mutated RB GeneScience, 1990
- The retinoblastoma protein is phosphorylated during specific phases of the cell cycleCell, 1989
- A novel human muscle factor related to but distinct from MyoD1 induces myogenic conversion in 10T1/2 fibroblasts.The EMBO Journal, 1989
- Cytoplasmic activation of human nuclear genes in stable heterocaryonsCell, 1983