Comparative Genomic Hybridization Identifies Loss of 18q22-qter as an Early and Specific Event in Tumorigenesis of Midgut Carcinoids
- 1 May 2001
- journal article
- research article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 158 (5), 1803-1808
- https://doi.org/10.1016/s0002-9440(10)64136-3
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Genomic Alterations in Well-Differentiated Gastrointestinal and Bronchial Neuroendocrine Tumors (Carcinoids): Marked Differences Indicating Diversity in Molecular PathogenesisThe American Journal of Pathology, 2000
- Mutations in SDHD , a Mitochondrial Complex II Gene, in Hereditary ParagangliomaScience, 2000
- Typical and Atypical Carcinoid Tumors of the Lung Are Characterized by 11q Deletions as Detected by Comparative Genomic HybridizationThe American Journal of Pathology, 1998
- Molecular Cloning and Characterization ofLOH11CR2A,a New Gene within a Refined Minimal Region of LOH at 11q23Genomics, 1997
- Identification of the multiple endocrine neoplasia type 1 (MEN1) gene. The European Consortium on MEN1Human Molecular Genetics, 1997
- A Human Gene (DDX10) Encoding a Putative DEAD-Box RNA Helicase at 11q22–q23Genomics, 1996
- DCC: linking tumor suppressor genes and altered cell surface interactions in cancer?Current Opinion in Genetics & Development, 1995
- Optimizing comparative genomic hybridization for analysis of DNA sequence copy number changes in solid tumorsGenes, Chromosomes and Cancer, 1994
- The DCC Gene: Structural Analysis and Mutations in Colorectal CarcinomasGenomics, 1994
- TP53 tumor suppressor gene: A model for investigating human mutagenesisGenes, Chromosomes and Cancer, 1992