Activation of JNK pathway in persistent pain
- 6 June 2008
- journal article
- review article
- Published by Elsevier BV in Neuroscience Letters
- Vol. 437 (3), 180-183
- https://doi.org/10.1016/j.neulet.2008.03.017
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Nitric oxide‐induced apoptosis in cultured rat astrocytes: Protection by edaravone, a radical scavengerGlia, 2007
- Activation of extracellular signal‐regulated protein kinases 5 in primary afferent neurons contributes to heat and cold hyperalgesia after inflammationJournal of Neurochemistry, 2007
- Lipoxins and aspirin-triggered lipoxin inhibit inflammatory pain processingThe Journal of Experimental Medicine, 2007
- Uses for JNK: the Many and Varied Substrates of the c-Jun N-Terminal KinasesMicrobiology and Molecular Biology Reviews, 2006
- Possible role of spinal astrocytes in maintaining chronic pain sensitization: review of current evidence with focus on bFGF/JNK pathwayNeuron Glia Biology, 2006
- c-Jun N-terminal kinase activation in dorsal root ganglion contributes to pain hypersensitivityBiochemical and Biophysical Research Communications, 2005
- The Invulnerability of Adult Neurons: A Critical Role for p73Journal of Neuroscience, 2004
- Reactive oxygen species (ROS) play an important role in a rat model of neuropathic painPain, 2004
- Prominent Expression of bFGF in Dorsal Root Ganglia after AxotomyEuropean Journal of Neuroscience, 1995
- Reactive astrogliosis after basic fibroblast growth factor (bFGF) injection in injured neonatal rat brainGlia, 1990