Matrix Metalloproteinase-1 and Thrombin Differentially Activate Gene Expression in Endothelial Cells via PAR-1 and Promote Angiogenesis
- 31 December 2008
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 173 (6), 1736-1746
- https://doi.org/10.2353/ajpath.2008.080512
Abstract
No abstract availableKeywords
Funding Information
- Norris Cotton Cancer Center (CA-77267, AR-26599, T32-AI07363)
- National Institutes of Health (CA-77267, AR-26599, T32-AI07363)
This publication has 61 references indexed in Scilit:
- Matrix metalloproteinase-1 promotes breast cancer angiogenesis and osteolysis in a novel in vivo modelBreast Cancer Research and Treatment, 2008
- Integrins in angiogenesis and lymphangiogenesisNature Reviews Cancer, 2008
- TNF primes endothelial cells for angiogenic sprouting by inducing a tip cell phenotypeBlood, 2008
- Inhibition of Angiogenesis by Small-Molecule Antagonists of Protease-Activated Receptor-1Seminars in Thrombosis and Hemostasis, 2007
- Matrix metalloproteinases and the regulation of tissue remodellingNature Reviews Molecular Cell Biology, 2007
- TUMOR STROMA AND REGULATION OF CANCER DEVELOPMENTAnnual Review Of Pathology-Mechanisms Of Disease, 2006
- Identification of membrane type-1 matrix metalloproteinase as a target of hypoxia-inducible factor-2α in von Hippel–Lindau renal cell carcinomaOncogene, 2004
- Role of Thrombin in Angiogenesis and Tumor ProgressionSeminars in Thrombosis and Hemostasis, 2004
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001
- Tumors: Wounds That Do Not HealNew England Journal of Medicine, 1986