Protein Kinase Cθ Negatively Regulates Store-independent Ca2+ Entry and Phosphatidylserine Exposure Downstream of Glycoprotein VI in Platelets
Open Access
- 25 June 2010
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 285 (26), 19865-19873
- https://doi.org/10.1074/jbc.m109.085654
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Rapid Procoagulant Phosphatidylserine Exposure Relies on High Cytosolic Calcium Rather Than on Mitochondrial DepolarizationArteriosclerosis, Thrombosis, and Vascular Biology, 2009
- A key role for dense granule secretion in potentiation of the Ca2+ signal arising from store-operated calcium entry in human plateletsCell Calcium, 2009
- Impaired activation of platelets lacking protein kinase C-θ isoformBlood, 2009
- PKCα regulates platelet granule secretion and thrombus formation in miceJCI Insight, 2009
- R93W mutation in Orai1 causes impaired calcium influx in plateletsBlood, 2009
- Genetic Analysis of the Role of Protein Kinase Cθ in Platelet Function and Thrombus FormationPLOS ONE, 2008
- Molecular and electrophysiological characterization of transient receptor potential ion channels in the primary murine megakaryocyteThe Journal of Physiology, 2006
- A role for PKCθ in outside‐in αIIbβ3 signalingJournal of Thrombosis and Haemostasis, 2006
- Store-Operated Calcium ChannelsPhysiological Reviews, 2005
- Exocytotic Insertion of TRPC6 Channel into the Plasma Membrane upon Gq Protein-coupled Receptor ActivationJournal of Biological Chemistry, 2004