Functional coupling of TRPM2 and extrasynaptic NMDARs exacerbates excitotoxicity in ischemic brain injury
- 12 April 2022
- journal article
- research article
- Published by Elsevier BV in Neuron
- Vol. 110 (12), 1944-+
- https://doi.org/10.1016/j.neuron.2022.03.021
Abstract
No abstract availableThis publication has 76 references indexed in Scilit:
- Redox signal-mediated sensitization of transient receptor potential melastatin 2 (TRPM2) to temperature affects macrophage functionsProceedings of the National Academy of Sciences of the United States of America, 2012
- The voltage-gated proton channel Hv1 enhances brain damage from ischemic strokeNature Neuroscience, 2012
- Sex Differences in Neuroprotection Provided by Inhibition of TRPM2 Channels following Experimental StrokeJournal of Cerebral Blood Flow & Metabolism, 2011
- State-dependent Inhibition of TRPM2 Channel by Acidic pHOnline Journal of Public Health Informatics, 2010
- Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disordersNature Reviews Neuroscience, 2010
- Structural Basis of Dimerization-dependent Ubiquitination by the SCFFbx4 Ubiquitin LigaseOnline Journal of Public Health Informatics, 2010
- CXCL12 inhibits expression of the NMDA receptor's NR2B subunit through a histone deacetylase-dependent pathway contributing to neuronal survivalCell Death & Disease, 2010
- Mitochondrial calcium and the permeability transition in cell deathBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2009
- Intracellular calcium activates TRPM2 and its alternative spliced isoformsProceedings of the National Academy of Sciences of the United States of America, 2009
- TRPM2-mediated Ca2+ influx induces chemokine production in monocytes that aggravates inflammatory neutrophil infiltrationNature Medicine, 2008