LPS-induced iNOS expression in Bv-2 cells is suppressed by an oxidative mechanism acting on the JNK pathway—A potential role for neuroprotection
- 31 March 2010
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1322, 1-7
- https://doi.org/10.1016/j.brainres.2010.01.082
Abstract
No abstract availableKeywords
Funding Information
- Estonian Ministry of Education and Science (SF0130171s08)
- Estonian Science Foundation (ETF7333, ETF7338, MJD7)
This publication has 31 references indexed in Scilit:
- Mechanisms of inflammatory neurodegeneration: iNOS and NADPH oxidaseBiochemical Society Transactions, 2007
- Reactive oxygen and nitrogen species as signaling molecules regulating neutrophil functionFree Radical Biology & Medicine, 2007
- Microglia-mediated neurotoxicity: uncovering the molecular mechanismsNature Reviews Neuroscience, 2007
- Potent regulation of microglia‐derived oxidative stress and dopaminergic neuron survival: substance P vs. dynorphinThe FASEB Journal, 2006
- Differential activation of astrocytes by innate and adaptive immune stimuliGlia, 2004
- Antioxidant Effects of Sulfur-Containing Amino AcidsYonsei Medical Journal, 2004
- Transforming Growth Factor-β1-Mediated Neuroprotection against Excitotoxic Injuryin VivoJournal of Cerebral Blood Flow & Metabolism, 2003
- Extracellular Signal-Regulated Kinase and p38 Subgroups of Mitogen-Activated Protein Kinases Regulate Inducible Nitric Oxide Synthase and Tumor Necrosis Factor-α Gene Expression in Endotoxin-Stimulated Primary Glial CulturesJournal of Neuroscience, 1998
- A femtomolar-acting neuroprotective peptide.JCI Insight, 1996
- Immortalization of murine microglial cells by a v-raf / v-myc carrying retrovirusJournal of Neuroimmunology, 1990