Two Divergent Signaling Pathways for TGF-β Separated by a Mutation of Its Type II Receptor Gene
- 7 June 1999
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 259 (2), 385-390
- https://doi.org/10.1006/bbrc.1999.0788
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 29 references indexed in Scilit:
- Transcriptional Activators of TGF-β Responses: SmadsCell, 1998
- The L45 loop in type I receptors for TGF‐β family members is a critical determinant in specifying Smad isoform activationFEBS Letters, 1998
- The tumor suppressor Smad4/DPC4 and transcriptional adaptor CBP/p300 are coactivators for Smad3 in TGF-β-induced transcriptional activationGenes & Development, 1998
- Determinants of specificity in TGF-β signal transductionGenes & Development, 1998
- HNPCC associated with germline mutation in the TGF-β type II receptor geneNature Genetics, 1998
- Repression of the CDK activator Cdc25A and cell-cycle arrest by cytokine TGF-β in cells lacking the CDK inhibitor p15Nature, 1997
- Identification of Important Regions in the Cytoplasmic Juxtamembrane Domain of Type I Receptor That Separate Signaling Pathways of Transforming Growth Factor-βOnline Journal of Public Health Informatics, 1996
- Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-beta.Genes & Development, 1995
- pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrestNature, 1994
- TGFβ signals through a heteromeric protein kinase receptor complexCell, 1992