RIP-Cre Revisited, Evidence for Impairments of Pancreatic β-Cell Function
Open Access
- 1 February 2006
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 281 (5), 2649-2653
- https://doi.org/10.1074/jbc.m512373200
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- The transcription factor Stat3 is dispensable for pancreatic β-cell development and functionBiochemical and Biophysical Research Communications, 2005
- SSTR5 ablation in islet results in alterations in glucose homeostasis in miceFEBS Letters, 2005
- Insulin secretory defects and impaired islet architecture in pancreatic β-cell-specific STAT3 knockout miceBiochemical and Biophysical Research Communications, 2004
- Conditional Inactivation of the Men1 Gene Leads to Pancreatic and Pituitary Tumorigenesis but Does Not Affect Normal Development of These TissuesMolecular and Cellular Biology, 2004
- Essential Role of STAT3 in Body Weight and Glucose HomeostasisMolecular and Cellular Biology, 2004
- Targeted Elimination of Peroxisome Proliferator-Activated Receptor γ in β Cells Leads to Abnormalities in Islet Mass without Compromising Glucose HomeostasisMolecular and Cellular Biology, 2003
- β-cell–specific deletion of the Igf1 receptor leads to hyperinsulinemia and glucose intolerance but does not alter β-cell massNature Genetics, 2002
- Tissue-specific deletion of Foxa2 in pancreatic β cells results in hyperinsulinemic hypoglycemiaGenes & Development, 2001
- Tissue-Specific Knockout of the Insulin Receptor in Pancreatic β Cells Creates an Insulin Secretory Defect Similar to that in Type 2 DiabetesCell, 1999
- Dual Roles for Glucokinase in Glucose Homeostasis as Determined by Liver and Pancreatic β Cell-specific Gene Knock-outs Using Cre RecombinaseJournal of Biological Chemistry, 1999