Hemodynamic consequences of atrial fibrillation at constant ventricular rates
- 1 March 1964
- journal article
- research article
- Published by Elsevier BV in American Journal Of Medicine
- Vol. 36 (3), 342-350
- https://doi.org/10.1016/0002-9343(64)90160-3
Abstract
Dogs which were areflexic and had induced heart block were studied and the consequences of atrial fibrillation at a constant, regular ventricular rate were examined. Alterations incurred by this arrhythmia could thus be related to the absence of effective atrial function per se. The observed hemodynamic consequences of atrial fibrillation were (1) an increase of mean left atrial pressure and an increase of mean left atrial pressure relative to left ventricular end-diastolic pressure; and (2) a decrease of aortic pressure and aortic flow. Evidence is presented which supports the view that the absence of effective atrial activity during atrial fibrillation results in early mitral regurgitation. Criteria for atrial fibrillation were also established by using 2 bipolar recording electrodes attached to the left atrium. These were lack of uniform polarity, lack of a phase relationship of the depolarization complex recorded at these 2 points, and irregular rate of depolarization at each recording site.Keywords
This publication has 30 references indexed in Scilit:
- The transport function of the atriumThe American Journal of Cardiology, 1962
- The Use of Ascorbate Dilution Curves in Cardiovascular DiagnosisCirculation, 1961
- Studies on Starling's Law of the HeartCirculation, 1961
- Detection of Right-to-Left Shunts with an Arterial Potentiometric ElectrodeCirculation, 1960
- Physiopathologic study (clinical and experimental) of the tricuspid valveThe American Journal of Cardiology, 1959
- Hemodynamic methods for differentiation of mitral stenosis and regurgitation∗The American Journal of Cardiology, 1958
- Cardiocirculatory Performance in Atrial FlutterCell Metabolism, 1955
- MITRAL INCOMPETENCE IN EXPERIMENTAL AURICULAR FIBRILLATIONThe Lancet, 1955
- The Diagnosis of Tricuspid InsufficiencyCell Metabolism, 1955
- TRICUSPID INCOMPETENCEHeart, 1954