p16INK4a and p14ARF Tumor Suppressor Genes Are Commonly Inactivated in Cutaneous Squamous Cell Carcinoma
- 1 May 2004
- journal article
- Published by Elsevier BV in Journal of Investigative Dermatology
- Vol. 122 (5), 1284-1292
- https://doi.org/10.1111/j.0022-202x.2004.22501.x
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- An Assessment of the CDKN2A Variant Ala148Thr as a Nevus/Melanoma Susceptibility AlleleJournal of Investigative Dermatology, 2002
- Prognostic significance of p16INK4a alterations and 9p21 loss of heterozigosity in locally advanced laryngeal squamous cell carcinomaJournal of Cellular Physiology, 2002
- Human papillomaviruses and non-melanoma skin cancerCurrent Opinion in Infectious Diseases, 2002
- Cutaneous Squamous-Cell CarcinomaNew England Journal of Medicine, 2001
- Changing trends in non‐melanoma skin cancer in South Wales, 1988–98British Journal of Dermatology, 2000
- CDKN2a/p16INK4a Mutations and Lack of p19ARF Involvement in Familial Melanoma KindredsJournal of Investigative Dermatology, 1998
- p14ARF links the tumour suppressors RB and p53Nature, 1998
- The Molecular Basis of Nonmelanoma Skin CancerArchives of Dermatology, 1997
- Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands.Proceedings of the National Academy of Sciences of the United States of America, 1996
- A role for sunlight in skin cancer: UV-induced p53 mutations in squamous cell carcinoma.Proceedings of the National Academy of Sciences, 1991