Blocking Intercellular Adhesion Molecule-1 on Human Epithelial Cells Decreases Respiratory Syncytial Virus Infection
- 1 January 2001
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 280 (1), 188-195
- https://doi.org/10.1006/bbrc.2000.4093
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Rhinovirus Infection Induces Expression of Its Own Receptor Intercellular Adhesion Molecule 1 (ICAM-1) via Increased NF-κB-mediated TranscriptionPublished by Elsevier BV ,1999
- Respiratory Syncytial Virus Induces the Expression of 5-Lipoxygenase and Endothelin-1 in Bronchial Epithelial CellsBiochemical and Biophysical Research Communications, 1998
- Respiratory Syncytial Virus Induces Selective Production of the Chemokine RANTES by Upper Airway Epithelial CellsThe Journal of Infectious Diseases, 1997
- Expression of Adhesion Molecules (ICAM-1, LFA-3) on Human Epithelial Cells (A549) after Respiratory Syncytial Virus InfectionInternational Archives of Allergy and Immunology, 1995
- Immunity to and Frequency of Reinfection with Respiratory Syncytial VirusThe Journal of Infectious Diseases, 1991
- Analysis of the local and systemic immune responses induced in BALB/c mice by experimental respiratory syncytial virus infectionJournal of General Virology, 1990
- A soluble form of intercellular adhesion molecule-1 inhibits rhinovirus infectionNature, 1990
- A cell adhesion molecule, ICAM-1, is the major surface receptor for rhinovirusesCell, 1989
- The major human rhinovirus receptor is ICAM-1Cell, 1989
- Functional evidence that intercellular adhesion molecule‐1 (icam‐1) is a ligand for lfa‐1d‐ependent adhesion in t cell‐mediated cytotoxicityEuropean Journal of Immunology, 1988