Persistently Activated Stat3 Maintains Constitutive NF-κB Activity in Tumors
Top Cited Papers
- 7 April 2009
- journal article
- research article
- Published by Elsevier BV in Cancer Cell
- Vol. 15 (4), 283-293
- https://doi.org/10.1016/j.ccr.2009.02.015
Abstract
No abstract availableThis publication has 46 references indexed in Scilit:
- IL-6 and Stat3 Are Required for Survival of Intestinal Epithelial Cells and Development of Colitis-Associated CancerCancer Cell, 2009
- Regulation of the IL-23 and IL-12 Balance by Stat3 Signaling in the Tumor MicroenvironmentCancer Cell, 2009
- Stat3 mediates myeloid cell–dependent tumor angiogenesis in miceJCI Insight, 2008
- Unphosphorylated STAT3 accumulates in response to IL-6 and activates transcription by binding to NFκBGenes & Development, 2007
- Crosstalk between cancer and immune cells: role of STAT3 in the tumour microenvironmentNature Reviews Immunology, 2007
- NF-κB: linking inflammation and immunity to cancer development and progressionNature Reviews Immunology, 2005
- Nuclear interaction of EGFR and STAT3 in the activation of the iNOS/NO pathwayCancer Cell, 2005
- The STATs of cancer — new molecular targets come of ageNature Reviews Cancer, 2004
- Secretion of cytokines and growth factors as a general cause of constitutive NFκB activation in cancerOncogene, 2003
- NF-κB in cancer: from innocent bystander to major culpritNature Reviews Cancer, 2002