Cdc42 Is a Rho GTPase Family Member That Can Mediate Insulin Signaling to Glucose Transport in 3T3-L1 Adipocytes
Open Access
- 1 April 2003
- journal article
- Published by Elsevier BV
- Vol. 278 (16), 13765-13774
- https://doi.org/10.1074/jbc.m208904200
Abstract
No abstract availableThis publication has 36 references indexed in Scilit:
- A Phosphatidylinositol 3-Kinase-independent Insulin Signaling Pathway to N-WASP/Arp2/3/F-actin Required for GLUT4 Glucose Transporter RecyclingPublished by Elsevier BV ,2002
- Gα11 Signaling through ARF6 Regulates F-Actin Mobilization and GLUT4 Glucose Transporter Translocation to the Plasma MembraneMolecular and Cellular Biology, 2001
- ADP-Ribosylation Factor 6 Delineates Separate Pathways Used by Endothelin 1 and Insulin for Stimulating Glucose Uptake in 3T3-L1 AdipocytesMolecular and Cellular Biology, 2001
- Rosiglitazone, Insulin Treatment, and Fasting Correct Defective Activation of Protein Kinase C- / by Insulin in Vastus Lateralis Muscles and Adipocytes of Diabetic RatsEndocrinology, 2001
- Overexpression of SH2-Containing Inositol Phosphatase 2 Results in Negative Regulation of Insulin-Induced Metabolic Actions in 3T3-L1 Adipocytes via Its 5′-Phosphatase Catalytic ActivityMolecular and Cellular Biology, 2001
- The osmotic shock-induced glucose transport pathway in 3T3-L1 adipocytes is mediated by Gab-1 and requires Gab-1 associated phosphatidylinositol 3-kinase activity for full activationJournal of Biological Chemistry, 2000
- The Trimeric GTP-binding Protein (Gq/G11) α Subunit Is Required for Insulin-stimulated GLUT4 Translocation in 3T3L1 AdipocytesPublished by Elsevier BV ,2000
- Endothelin-1-induced GLUT4 Translocation Is Mediated via Gαq/11 Protein and Phosphatidylinositol 3-Kinase in 3T3-L1 AdipocytesPublished by Elsevier BV ,1999
- Stimulation of IRS-1-associated Phosphatidylinositol 3-Kinase and Akt/Protein Kinase B but Not Glucose Transport by β1-Integrin Signaling in Rat AdipocytesPublished by Elsevier BV ,1998
- Insulin-stimulated GLUT4 Translocation Is Mediated by a Divergent Intracellular Signaling PathwayPublished by Elsevier BV ,1995