Genetic variants at the 9p21 locus contribute to atherosclerosis through modulation of ANRIL and CDKN2A/B
Top Cited Papers
- 29 February 2012
- journal article
- Published by Elsevier BV in Atherosclerosis
- Vol. 220 (2), 449-455
- https://doi.org/10.1016/j.atherosclerosis.2011.11.017
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Long non-coding RNA ANRIL is required for the PRC2 recruitment to and silencing of p15INK4B tumor suppressor geneOncogene, 2010
- Associations of 9p21 variants with cutaneous malignant melanoma, nevi, and pigmentation phenotypes in melanoma-prone families with and without CDKN2A mutationsFamilial Cancer, 2010
- Molecular Interplay of the Noncoding RNA ANRIL and Methylated Histone H3 Lysine 27 by Polycomb CBX7 in Transcriptional Silencing of INK4aMolecular Cell, 2010
- Variation in CDKN2A at 9p21.3 influences childhood acute lymphoblastic leukemia riskNature Genetics, 2010
- Genome-wide association study identifies variants at 9p21 and 22q13 associated with development of cutaneous neviNature Genetics, 2009
- Sequence variants on chromosome 9p21.3 confer risk for atherosclerotic strokeAnnals of Neurology, 2009
- The same sequence variant on 9p21 associates with myocardial infarction, abdominal aortic aneurysm and intracranial aneurysmNature Genetics, 2008
- Susceptibility to coronary artery disease and diabetes is encoded by distinct, tightly linked SNPs in the ANRIL locus on chromosome 9pHuman Molecular Genetics, 2007
- Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controlsNature, 2007
- Regulation of the INK4b–ARF–INK4a tumour suppressor locus: all for one or one for allNature Reviews Molecular Cell Biology, 2006