Follistatin-like 1, a Secreted Muscle Protein, Promotes Endothelial Cell Function and Revascularization in Ischemic Tissue through a Nitric-oxide Synthase-dependent Mechanism
Open Access
- 1 November 2008
- journal article
- Published by Elsevier BV
- Vol. 283 (47), 32802-32811
- https://doi.org/10.1074/jbc.m803440200
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Follistatin-Like 1 Is an Akt-Regulated Cardioprotective Factor That Is Secreted by the HeartCirculation, 2008
- Fast/Glycolytic Muscle Fiber Growth Reduces Fat Mass and Improves Metabolic Parameters in Obese MiceCell Metabolism, 2008
- Vascular Endothelial Growth Factor Blockade Promotes the Transition From Compensatory Cardiac Hypertrophy to Failure in Response to Pressure OverloadHypertension, 2006
- Angiogenesis in life, disease and medicineNature, 2005
- Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failureJCI Insight, 2005
- AMP-Activated Protein Kinase Signaling Stimulates VEGF Expression and Angiogenesis in Skeletal MuscleCirculation Research, 2005
- Conditional Activation of Akt in Adult Skeletal Muscle Induces Rapid HypertrophyMolecular and Cellular Biology, 2004
- Regulation of Angiogenesis by Glycogen Synthase Kinase-3βPublished by Elsevier BV ,2002
- Regulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasionOncogene, 2000
- Cloning from a mouse osteoblastic cell line of a set of transforming‐growth‐factor‐β1‐regulated genes, one of which seems to encode a follistatin‐related polypeptideJBIC Journal of Biological Inorganic Chemistry, 1993