PPARs are a unique set of fatty acid regulated transcription factors controlling both lipid metabolism and inflammation
Top Cited Papers
- 31 August 2011
- journal article
- review article
- Published by Elsevier BV in Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
- Vol. 1812 (8), 1007-1022
- https://doi.org/10.1016/j.bbadis.2011.02.014
Abstract
No abstract availableKeywords
Funding Information
- Hungarian Scientific Research Fund (NK72730)
This publication has 169 references indexed in Scilit:
- STAT6 Transcription Factor Is a Facilitator of the Nuclear Receptor PPARγ-Regulated Gene Expression in Macrophages and Dendritic CellsImmunity, 2010
- Anti-diabetic drugs inhibit obesity-linked phosphorylation of PPARγ by Cdk5Nature, 2010
- Alternative Activation of Macrophages: Mechanism and FunctionsImmunity, 2010
- Murine atopic dermatitis responds to peroxisome proliferator-activated receptors α and β/δ (but not γ) and liver X receptor activatorsJournal of Allergy and Clinical Immunology, 2010
- PPAR-δ senses and orchestrates clearance of apoptotic cells to promote toleranceNature Medicine, 2009
- Adipocyte-Derived Th2 Cytokines and Myeloid PPARδ Regulate Macrophage Polarization and Insulin SensitivityCell Metabolism, 2008
- Macrophage-specific PPARγ controls alternative activation and improves insulin resistanceNature, 2007
- Inflammation and metabolic disordersNature, 2006
- Non-DNA binding, dominant-negative, human PPARγ mutations cause lipodystrophic insulin resistanceCell Metabolism, 2006
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994