Lesion of the anteroventral third ventricle (AV3V) reduces hypothalamic activation and hypophyseal hormone secretion induced by lipopolysaccharide in rats
- 18 October 2006
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1115 (1), 83-91
- https://doi.org/10.1016/j.brainres.2006.07.087
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- Septic shockThe Lancet, 2005
- Plasma Interleukin-1β, Prolactin, ACTH and Corticosterone Responses to Endotoxin after Damage of the Anterior Hypothalamic AreaNeuroimmunomodulation, 2001
- Evidence for the involvement of AV3V in the circulating IL-1β-to-brain communicationJournal of Thermal Biology, 2000
- Anteroventral Third Ventricle Lesion Suppresses Fever, but Not Stress‐Induced Hyperthermia in RatsaAnnals of the New York Academy of Sciences, 1997
- The Hypothalamic–Pituitary–Adrenal Axis and Immune-Mediated InflammationThe New England Journal of Medicine, 1995
- Role of Cytokines in the Endocrine SystemAnnals of the New York Academy of Sciences, 1994
- Ablation of the region anterior and ventral to the third ventricle (AV3V region) does not impede parturition in ratsJournal of Endocrinology, 1989
- Baroreceptor influences on oxytocin and vasopressin secretion.Hypertension, 1989
- Control of magnocellular oxytocin neurones by the region anterior and ventral to the third ventricle (AV3V region) in ratsJournal of Endocrinology, 1987
- Changes in Magnocellular-Neurohypophyseal Vasopressin Following Anteroventral Third-Ventricle (AV3V) LesionsJournal of Cardiovascular Pharmacology, 1986