Strain influences on inflammatory pathway activation, cell infiltration and complement cascade after traumatic brain injury in the rat
- 1 January 2013
- journal article
- research article
- Published by Elsevier BV in Brain, Behavior, and Immunity
- Vol. 27 (1), 109-122
- https://doi.org/10.1016/j.bbi.2012.10.002
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- A Novel NKR-P1Bbright NK Cell Subset Expresses an Activated CD25+CX3CR1+CD62L−CD11b−CD27− Phenotype and Is Prevalent in Blood, Liver, and Gut-Associated Lymphoid Organs of RatsThe Journal of Immunology, 2012
- Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in miceJournal of Neuroinflammation, 2012
- The cytokine temporal profile in rat cortex after controlled cortical impactFrontiers in Molecular Neuroscience, 2012
- Inflammation after trauma: Microglial activation and traumatic brain injuryAnnals of Neurology, 2011
- Strain-Related Differences after Experimental Traumatic Brain Injury in RatsJournal of Neurotrauma, 2010
- Blood–brain barrier breakdown as a therapeutic target in traumatic brain injuryNature Reviews Neurology, 2010
- Multiple loci comprising immune-related genes regulate experimental neuroinflammationGenes & Immunity, 2009
- The Role of the Choroid Plexus in Neutrophil Invasion after Traumatic Brain InjuryJournal of Cerebral Blood Flow & Metabolism, 2009
- The determinants of susceptibility/resistance to adjuvant arthritis in ratsArthritis Research & Therapy, 2009
- Complement regulator loss on apoptotic neuronal cells causes increased complement activation and promotes both phagocytosis and cell lysisMolecular Immunology, 2006