Multiplicative interactions to enhance gabapentin to treat neuropathic pain
- 19 November 2008
- journal article
- Published by Elsevier BV in European Journal of Pharmacology
- Vol. 598 (1-3), 21-26
- https://doi.org/10.1016/j.ejphar.2008.09.004
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Calcium channel α2δ1 subunit mediates spinal hyperexcitability in pain modulationPain, 2006
- Depletion of endogenous spinal 5-HT attenuates the behavioural hypersensitivity to mechanical and cooling stimuli induced by spinal nerve ligationPain, 2006
- The Monoamine-Mediated Antiallodynic Effects of Intrathecally Administered Milnacipran, a Serotonin Noradrenaline Reuptake Inhibitor, in a Rat Model of Neuropathic PainAnesthesia & Analgesia, 2005
- Bad news from the brain: descending 5-HT pathways that control spinal pain processingTrends in Pharmacological Sciences, 2004
- Treatment of opiate-related sedation: utility of the cholinesterase inhibitors.2004
- Efficacy of Duloxetine, a Potent and Balanced Serotonin-Norepinephrine Reuptake Inhibitor in Persistent Pain Models in RatsJournal of Pharmacology and Experimental Therapeutics, 2004
- Chronic pain and medullary descending facilitationTrends in Neurosciences, 2002
- Use of Gabapentin in the Treatment of Neuropathic PainAnnals of Pharmacotherapy, 2000
- Quantitative assessment of tactile allodynia in the rat pawJournal of Neuroscience Methods, 1994
- Statistical analysis of drug-drug and site-site interactions with isobologramsLife Sciences, 1989