S100B-stimulated NO production by BV-2 microglia is independent of RAGE transducing activity but dependent on RAGE extracellular domain
- 25 September 2004
- journal article
- review article
- Published by Elsevier BV in Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Vol. 1742 (1-3), 169-177
- https://doi.org/10.1016/j.bbamcr.2004.09.008
Abstract
No abstract availableThis publication has 36 references indexed in Scilit:
- Amphoterin Stimulates Myogenesis and Counteracts the Antimyogenic Factors Basic Fibroblast Growth Factor and S100B via RAGE BindingMolecular and Cellular Biology, 2004
- S100P Stimulates Cell Proliferation and Survival via Receptor for Activated Glycation End Products (RAGE)Published by Elsevier BV ,2004
- S100B Inhibits Myogenic Differentiation and Myotube Formation in a RAGE-Independent MannerMolecular and Cellular Biology, 2003
- NF-κB activation and IκBα dynamism involved in iNOS and chemokine induction in astroglial cellsLife Sciences, 2001
- Activation of A1 adenosine or mGlu3 metabotropic glutamate receptors enhances the release of nerve growth factor and S‐100β protein from cultured astrocytesGlia, 1999
- Receptor for Advanced Glycation End Products (RAGE)-mediated Neurite Outgrowth and Activation of NF-κB Require the Cytoplasmic Domain of the Receptor but Different Downstream Signaling PathwaysPublished by Elsevier BV ,1999
- RAGE and amyloid-β peptide neurotoxicity in Alzheimer's diseaseNature, 1996
- S100β protects hippocampal neurons from damage induced by glucose deprivationBrain Research, 1995
- An immortalized cell line expresses properties of activated microglial cellsJournal of Neuroscience Research, 1992
- Immortalization of murine microglial cells by a v-raf / v-myc carrying retrovirusJournal of Neuroimmunology, 1990