Impaired Nutrient Signaling and Body Weight Control in a Na+ Neutral Amino Acid Cotransporter (Slc6a19)-deficient Mouse
Open Access
- 1 July 2011
- journal article
- research article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 286 (30), 26638-26651
- https://doi.org/10.1074/jbc.m111.241323
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Loss-of-function mutations in the glutamate transporter SLC1A1 cause human dicarboxylic aminoaciduriaJCI Insight, 2011
- Renal imino acid and glycine transport system ontogeny and involvement in developmental iminoglycinuriaBiochemical Journal, 2010
- ATF4-dependent transcription mediates signaling of amino acid limitationTrends in Endocrinology & Metabolism, 2009
- Physiology and Function of the Tight JunctionCold Spring Harbor Perspectives in Biology, 2009
- Orphan Transporter SLC6A18 Is Renal Neutral Amino Acid Transporter B0AT3Published by Elsevier BV ,2009
- Iminoglycinuria and hyperglycinuria are discrete human phenotypes resulting from complex mutations in proline and glycine transportersJCI Insight, 2008
- Deletion of Nicotinamide Nucleotide TranshydrogenaseDiabetes, 2006
- Hypertension and Impaired Glycine Handling in Mice Lacking the Orphan Transporter XT2Molecular and Cellular Biology, 2004
- Multiple transport pathways for neutral amino acids in rabbit jejunal brush border vesiclesThe Journal of Membrane Biology, 1982
- Intestinal absorption of amino acids and peptidesProceedings of the Nutrition Society, 1972