Essential Roles for GSK-3s and GSK-3-Primed Substrates in Neurotrophin-Induced and Hippocampal Axon Growth
Open Access
- 1 December 2006
- journal article
- research article
- Published by Elsevier BV in Neuron
- Vol. 52 (6), 981-996
- https://doi.org/10.1016/j.neuron.2006.10.031
Abstract
No abstract availableFunding Information
- National Institutes of Health (R01NS050968)
This publication has 49 references indexed in Scilit:
- Presenilin‐1 is an unprimed glycogen synthase kinase‐3β substrateFEBS Letters, 2006
- Leptin Increases Axonal Growth Cone Size in Developing Mouse Cortical Neurons by Convergent Signals Inactivating Glycogen Synthase Kinase-3βPublished by Elsevier BV ,2006
- PTEN couples Sema3A signalling to growth cone collapseJournal of Cell Science, 2006
- Both the Establishment and the Maintenance of Neuronal Polarity Require Active Mechanisms: Critical Roles of GSK-3β and Its Upstream RegulatorsCell, 2005
- APC and GSK-3β Are Involved in mPar3 Targeting to the Nascent Axon and Establishment of Neuronal PolarityCurrent Biology, 2004
- Inhibition of glycogen synthase kinase 3β in sensory neurons in culture alters filopodia dynamics and microtubule distribution in growth conesMolecular and Cellular Neuroscience, 2003
- Insulin Mimetic Action of Synthetic Phosphorylated Peptide Inhibitors of Glycogen Synthase Kinase-3The Journal of pharmacology and experimental therapeutics, 2003
- CRMP-2 binds to tubulin heterodimers to promote microtubule assemblyNature, 2002
- A variant of yellow fluorescent protein with fast and efficient maturation for cell-biological applicationsNature Biotechnology, 2002
- Death of some dorsal root ganglion neurons and plasticity of others following sciatic nerve section in adult and neonatal ratsJournal of Comparative Neurology, 1989