TRAIL+ NK Cells Control CD4+ T Cell Responses during Chronic Viral Infection to Limit Autoimmunity
- 1 October 2014
- journal article
- research article
- Published by Elsevier BV in Immunity
- Vol. 41 (4), 646-656
- https://doi.org/10.1016/j.immuni.2014.09.013
Abstract
No abstract availableThis publication has 42 references indexed in Scilit:
- Yet another role for natural killer cells: Cytotoxicity in immune regulation and viral persistenceProceedings of the National Academy of Sciences of the United States of America, 2012
- Natural killer cell activation enhances immune pathology and promotes chronic infection by limiting CD8 + T-cell immunityProceedings of the National Academy of Sciences, 2011
- Natural killer cells act as rheostats modulating antiviral T cellsNature, 2011
- TNF-related apoptosis-inducing ligand (TRAIL) regulates inflammatory neutrophil apoptosis and enhances resolution of inflammationJournal of Leukocyte Biology, 2011
- Absence of mouse 2B4 promotes NK cell–mediated killing of activated CD8+ T cells, leading to prolonged viral persistence and altered pathogenesisJCI Insight, 2010
- Innate immunity defines the capacity of antiviral T cells to limit persistent infectionThe Journal of Experimental Medicine, 2010
- Activating receptors promote NK cell expansion for maintenance, IL-10 production, and CD8 T cell regulation during viral infectionThe Journal of Experimental Medicine, 2009
- Following TRAIL’s path in the immune systemImmunology, 2009
- Up on the tightrope: natural killer cell activation and inhibitionNature Immunology, 2008
- TRAIL limits excessive host immune responses in bacterial meningitisJCI Insight, 2007