Role of epithelial-to-mesenchymal transition (EMT) in drug sensitivity and metastasis in bladder cancer
Open Access
- 14 December 2009
- journal article
- review article
- Published by Springer Science and Business Media LLC in Cancer and Metastasis Reviews
- Vol. 28 (3), 335-344
- https://doi.org/10.1007/s10555-009-9194-7
Abstract
No abstract availableKeywords
This publication has 91 references indexed in Scilit:
- Contextual extracellular cues promote tumor cell EMT and metastasis by regulating miR-200 family expressionGenes & Development, 2009
- SIP1 protein protects cells from DNA damage-induced apoptosis and has independent prognostic value in bladder cancerProceedings of the National Academy of Sciences of the United States of America, 2009
- Role of neutrophils in BCG immunotherapy for bladder cancerUrologic Oncology, 2008
- SMAD proteins control DROSHA-mediated microRNA maturationNature, 2008
- The Epithelial-Mesenchymal Transition Generates Cells with Properties of Stem CellsCell, 2008
- The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2Genes & Development, 2008
- Death-receptor O-glycosylation controls tumor-cell sensitivity to the proapoptotic ligand Apo2L/TRAILNature Medicine, 2007
- FGFR3 protein expression and its relationship to mutation status and prognostic variables in bladder cancerThe Journal of Pathology, 2007
- Quantitative analysis of EGFRvIII cellular signaling networks reveals a combinatorial therapeutic strategy for glioblastomaProceedings of the National Academy of Sciences of the United States of America, 2007
- Knockdown by shRNA identifies S249C mutant FGFR3 as a potential therapeutic target in bladder cancerOncogene, 2007