Oxidative-stress-induced alterations in Sp factors mediate transcriptional regulation of the NR1 subunit in hippocampus during hypoxia
- 15 July 2010
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 49 (2), 178-191
- https://doi.org/10.1016/j.freeradbiomed.2010.03.027
Abstract
No abstract availableKeywords
This publication has 59 references indexed in Scilit:
- Glutamate‐mediated excitotoxicity in neonatal hippocampal neurons is mediated by mGluR‐induced release of Ca++ from intracellular stores and is prevented by estradiolEuropean Journal of Neuroscience, 2006
- Glutamate receptor activation evokes calpain-mediated degradation of Sp3 and Sp4, the prominent Sp-family transcription factors in neuronsJournal of Neurochemistry, 2006
- NR2B Subunit Exerts a Critical Role in Postischemic Synaptic PlasticityStroke, 2006
- Dystrophin Dp71 Expression Is Down-regulated during MyogenesisOnline Journal of Public Health Informatics, 2005
- Estrogen regulation of trefoil factor 1 expression by estrogen receptor α and Sp proteinsExperimental Cell Research, 2005
- Proteasomal dysfunction induced by 4‐hydroxy‐2,3‐trans‐nonenal, an end‐product of lipid peroxidation: a mechanism contributing to neurodegeneration?Journal of Neurochemistry, 2002
- Oxidative Stress-Associated Impairment of Proteasome Activity during Ischemia–Reperfusion InjuryJournal of Cerebral Blood Flow & Metabolism, 2000
- Geldanamycin, an inhibitor of heat shock protein 90 (Hsp90) mediated signal transduction has anti‐inflammatory effects and interacts with glucocorticoid receptor in vivoBritish Journal of Pharmacology, 2000
- Hypoxia Regulates β-Enolase and Pyruvate Kinase-M Promoters by Modulating Sp1/Sp3 Binding to a Conserved GC ElementOnline Journal of Public Health Informatics, 1998
- Morphological alterations in the hippocampus following hypobaric hypoxiaHuman & Experimental Toxicology, 1996