Constitutive Activation of JAK3/STAT3 in Colon Carcinoma Tumors and Cell Lines: Inhibition of JAK3/STAT3 Signaling Induces Apoptosis and Cell Cycle Arrest of Colon Carcinoma Cells
- 31 October 2005
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 167 (4), 969-980
- https://doi.org/10.1016/s0002-9440(10)61187-x
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health
This publication has 55 references indexed in Scilit:
- STAT proteins: From normal control of cellular events to tumorigenesisJournal of Cellular Physiology, 2003
- Cancer Statistics, 2003CA: A Cancer Journal for Clinicians, 2003
- Src activation regulates anoikis in human colon tumor cell linesOncogene, 2002
- STAT-signalling through the cytoplasmic compartment:: Consideration of a new paradigm☆Cellular Signalling, 2000
- Bcl‐XL Antisense Sensitizes Human Colon Cancer Cell Line to 5‐FluorouracilJapanese Journal of Cancer Research, 2000
- Full Oncogenic Activities of v-Src Are Mediated by Multiple Signaling PathwaysOnline Journal of Public Health Informatics, 2000
- The role of STATs in transcriptional control and their impact on cellular functionOncogene, 2000
- Angiopoietin-1 Inhibits Endothelial Cell Apoptosis via the Akt/Survivin PathwayOnline Journal of Public Health Informatics, 2000
- Receptors for Interleukin (IL)-4 Do Not Associate with the Common γChain, and IL-4 Induces the Phosphorylation of JAK2 Tyrosine Kinase in Human Colon Carcinoma CellsOnline Journal of Public Health Informatics, 1995
- A Kinase-deficient Splice Variant of the Human JAK3 Is Expressed in Hematopoietic and Epithelial Cancer CellsOnline Journal of Public Health Informatics, 1995