Reply to: 'Clearance of amyloid β-peptide from brain: transport or metabolism?'
- 1 July 2000
- journal article
- Published by Springer Science and Business Media LLC in Nature Medicine
- Vol. 6 (7), 718-719
- https://doi.org/10.1038/77399
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Identification of the major Aβ1–42-degrading catabolic pathway in brain parenchyma: Suppression leads to biochemical and pathological depositionNature Medicine, 2000
- Human blood-brain barrier receptors for Alzheimer's amyloid-beta 1- 40. Asymmetrical binding, endocytosis, and transcytosis at the apical side of brain microvascular endothelial cell monolayer.JCI Insight, 1998
- α2‐Macroglobulin Complexes with and Mediates the Endocytosis of β‐Amyloid Peptide via Cell Surface Low‐Density Lipoprotein Receptor‐Related ProteinJournal of Neurochemistry, 1997
- Amyloid, the presenilins and Alzheimer's diseaseTrends in Neurosciences, 1997
- Amyloid β‐Peptide Induces Cell Monolayer Albumin Permeability, Impairs Glucose Transport, and Induces Apoptosis in Vascular Endothelial CellsJournal of Neurochemistry, 1997
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic MiceScience, 1996
- RAGE and amyloid-β peptide neurotoxicity in Alzheimer's diseaseNature, 1996
- Glycoprotein 330/megalin: probable role in receptor-mediated transport of apolipoprotein J alone and in a complex with Alzheimer disease amyloid beta at the blood-brain and blood-cerebrospinal fluid barriers.Proceedings of the National Academy of Sciences of the United States of America, 1996
- β-Amyloid-mediated vasoactivity and vascular endothelial damageNature, 1996
- Seeding “one-dimensional crystallization” of amyloid: A pathogenic mechanism in Alzheimer's disease and scrapie?Cell, 1993