Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K+ current
- 1 November 2006
- journal article
- Published by Elsevier BV in Neurobiology of Aging
- Vol. 27 (11), 1673-1683
- https://doi.org/10.1016/j.neurobiolaging.2005.09.038
Abstract
No abstract availableKeywords
Funding Information
- Medical Research Council
- Alzheimer's Research Trust
- Alzheimer's Society
- Wellcome Trust
This publication has 32 references indexed in Scilit:
- A Cell Biological Perspective on Alzheimer's DiseaseAnnual Review of Cell and Developmental Biology, 2002
- Modulation of Kv4-encoded K+ Currents in the Mammalian Myocardium by Neuronal Calcium Sensor-1Journal of Biological Chemistry, 2002
- Amyloid ? protein is internalized selectively by hippocampal field CA1 and causes neurons to accumulate amyloidogenic carboxyterminal fragments of the amyloid precursor proteinJournal of Comparative Neurology, 1998
- Role of Potassium Channels in Amyloid‐Induced Cell DeathJournal of Neurochemistry, 1998
- Preferential adsorption, internalization and resistance to degradation of the major isoform of the Alzheimer's amyloid peptide, Aβ1–42, in differentiated PC12 cellsBrain Research, 1997
- Soluble β-amyloid induces Alzheimer's disease features in human fibroblasts and in neuronal tissuesLife Sciences, 1996
- Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neuronsBiophysical Journal, 1996
- Activation of K+ channels and suppression of neuronal activity by secreted β-amyloid-precursor proteinNature, 1996
- The brain Kv1.1 potassium channel: in vitro and in vivo studies on subunit assembly and posttranslational processingJournal of Neuroscience, 1994
- Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid proteinBiochemical and Biophysical Research Communications, 1984