Ablation of CD11c-Positive Cells Normalizes Insulin Sensitivity in Obese Insulin Resistant Animals
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Open Access
- 8 October 2008
- journal article
- research article
- Published by Elsevier BV in Cell Metabolism
- Vol. 8 (4), 301-309
- https://doi.org/10.1016/j.cmet.2008.08.015
Abstract
No abstract availableThis publication has 33 references indexed in Scilit:
- JNK1 in Hematopoietically Derived Cells Contributes to Diet-Induced Inflammation and Insulin Resistance without Affecting ObesityCell Metabolism, 2007
- Deficiency of Cbl-b Gene Enhances Infiltration and Activation of Macrophages in Adipose Tissue and Causes Peripheral Insulin Resistance in MiceDiabetes, 2007
- Osteopontin mediates obesity-induced adipose tissue macrophage infiltration and insulin resistance in miceJCI Insight, 2007
- Macrophage PPARγ is required for normal skeletal muscle and hepatic insulin sensitivity and full antidiabetic effects of thiazolidinedionesJCI Insight, 2007
- Obesity induces a phenotypic switch in adipose tissue macrophage polarizationJCI Insight, 2007
- MCP-1 contributes to macrophage infiltration into adipose tissue, insulin resistance, and hepatic steatosis in obesityJCI Insight, 2006
- CCR2 modulates inflammatory and metabolic effects of high-fat feedingJCI Insight, 2006
- Inflamed fat: what starts the fire?JCI Insight, 2005
- Monocyte and macrophage heterogeneityNature Reviews Immunology, 2005
- Peroxisome-proliferator-activated receptor-α (PPARα) deficiency leads to dysregulation of hepatic lipid and carbohydrate metabolism by fatty acids and insulinBiochemical Journal, 2002