Zinc inhibition of GABA-stimulated Cl− influx in rat brain regions is unaffected by acute or chronic benzodiazepine
- 30 November 1994
- journal article
- Published by Elsevier BV in Pharmacology Biochemistry and Behavior
- Vol. 49 (3), 477-482
- https://doi.org/10.1016/0091-3057(94)90058-2
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Tolerance to the effects of diazepam, clonazepam and bretazenil on GABA-stimulated Cl− influx in flurazepam tolerant ratsEuropean Journal of Pharmacology: Molecular Pharmacology, 1993
- Function and pharmacology of multiple GABAA receptor subunitsTrends in Pharmacological Sciences, 1991
- GABA A receptor subtypes: from pharmacology to molecular biologyThe FASEB Journal, 1991
- Decreased GABAA receptor subunit mRNA concentrations following chronic lorazepam administrationBritish Journal of Pharmacology, 1991
- Functional and molecular distinction between recombinant rat GABAA receptor subtypes by Zn2+Neuron, 1990
- Novel GABAA receptor α subunit is expressed only in cerebellar granule cellsJournal of Molecular Biology, 1990
- Effects of continuous diazepam administration on GABAA subunit mRNA in rat brainJournal of Molecular Neuroscience, 1990
- Structural and functional basis for GABAA receptor heterogeneityNature, 1988
- Divalent cations reduce depolarization of primary afferent terminations by GABABrain Research, 1987
- Characterization of GAVAA receptor-mediated 36chloride uptake in rat brain synaptoneurosomesLife Sciences, 1987