Interleukin-12 p40 gene (IL12B) 3′-untranslated region polymorphism is associated with susceptibility to atopic dermatitis and psoriasis vulgaris
- 1 November 2002
- journal article
- Published by Elsevier BV in Journal of Dermatological Science
- Vol. 30 (2), 161-166
- https://doi.org/10.1016/s0923-1811(02)00072-5
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Identification of missense mutation in the IL12B gene: lack of association between IL12B polymorphisms and asthma and allergic rhinitis in the Japanese populationGenes & Immunity, 2001
- Identifying genes predisposing to atopic eczemaJournal of Allergy and Clinical Immunology, 1999
- Linkage and association of an interleukin 4 gene polymorphism with atopic dermatitis in Japanese families.Journal of Medical Genetics, 1998
- The role of IL-12 in the induction of organ-specific autoimmune diseasesImmunology Today, 1995
- High-Resolution Physical Mapping of Human 5q31-q33 Using Three Methods: Radiation Hybrid Mapping, Interphase Fluorescence in Situ Hybridization, and Pulsed-Field Gel ElectrophoresisGenomics, 1994
- Differential expression of mRNA encoding interleukin‐12 p35 and p40 subunits in situEuropean Journal of Immunology, 1994
- Differential in situ cytokine gene expression in acute versus chronic atopic dermatitis.JCI Insight, 1994
- Lesional expression of interferon-γ in atopic eczemaThe Lancet, 1994
- Interleukin‐12/T cell stimulating factor, a cytokine with multiple effects on T helper type 1 (Th1) but not on Th2 cellsEuropean Journal of Immunology, 1993
- Natural killer cell stimulatory factor (interleukin 12 [IL-12]) induces T helper type 1 (Th1)-specific immune responses and inhibits the development of IL-4-producing Th cells.The Journal of Experimental Medicine, 1993