IgM monomers accelerate disease manifestations in autoimmune-prone Fas-deficient mice
- 31 December 2004
- journal article
- Published by Elsevier BV in Journal of Autoimmunity
- Vol. 23 (4), 333-343
- https://doi.org/10.1016/j.jaut.2004.09.001
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- I-PLA2 Activation during Apoptosis Promotes the Exposure of Membrane Lysophosphatidylcholine Leading to Binding by Natural Immunoglobulin M Antibodies and Complement ActivationThe Journal of Experimental Medicine, 2002
- Late events in assembly determine the polymeric structure and biological activity of secretory IgMMolecular Immunology, 1997
- B Cell Development under the Condition of Allelic InclusionImmunity, 1997
- Low molecular weight IgM in healthy adults: Influence of HLAHuman Immunology, 1996
- Complement deficiency and diseaseImmunology Today, 1991
- Lpr and gld: Single Gene Models of Systemic Autoimmunity and Lymphoproliferative DiseaseAnnual Review of Immunology, 1991
- Anti-DNA antibodies from autoimmune mice arise by clonal expansion and somatic mutation.The Journal of Experimental Medicine, 1990
- IgM - molecular requirements for its assembly and functionImmunology Today, 1989
- Feedback inhibition of immunoglobulin gene rearrangement by membrane mu, but not by secreted mu heavy chains.The Journal of Experimental Medicine, 1988
- Low molecular weight IgM in rheumatoid arthritis and other rheumatic diseasesArthritis & Rheumatism, 1981