Genomic analysis of marginal zone and lymphoplasmacytic lymphomas identified common and disease-specific abnormalities
Open Access
- 1 May 2012
- journal article
- research article
- Published by Elsevier BV in Laboratory Investigation
- Vol. 25 (5), 651-660
- https://doi.org/10.1038/modpathol.2011.213
Abstract
No abstract availableThis publication has 45 references indexed in Scilit:
- Progressive but previously untreated CLL patients with greater array CGH complexity exhibit a less durable response to chemoimmunotherapyCancer Genetics and Cytogenetics, 2010
- Pathway discovery in mantle cell lymphoma by integrated analysis of high-resolution gene expression and copy number profilingBlood, 2010
- The NF-κB negative regulator TNFAIP3 (A20) is inactivated by somatic mutations and genomic deletions in marginal zone lymphomasBlood, 2009
- Mutations of multiple genes cause deregulation of NF-κB in diffuse large B-cell lymphomaNature, 2009
- TNFAIP3 (A20) is a tumor suppressor gene in Hodgkin lymphoma and primary mediastinal B cell lymphomaThe Journal of Experimental Medicine, 2009
- Identification of Copy Number Abnormalities and Inactivating Mutations in Two Negative Regulators of Nuclear Factor-κB Signaling Pathways in Waldenström's MacroglobulinemiaCancer Research, 2009
- Gene expression profiling of pulmonary mucosa-associated lymphoid tissue lymphoma identifies new biologic insights with potential diagnostic and therapeutic applicationsBlood, 2009
- Molecular subtypes of diffuse large B-cell lymphoma arise by distinct genetic pathwaysProceedings of the National Academy of Sciences of the United States of America, 2008
- Promiscuous Mutations Activate the Noncanonical NF-κB Pathway in Multiple MyelomaCancer Cell, 2007
- Clinicopathological definition of Waldenstrom's macroglobulinemia: Consensus Panel Recommendations from the Second International Workshop on Waldenstrom's MacroglobulinemiaSeminars in Oncology, 2003