Hepatitis C virus expression and interferon antiviral action is dependent on PKR expression
- 27 June 2007
- journal article
- research article
- Published by Wiley in Journal of Medical Virology
- Vol. 79 (8), 1120-1127
- https://doi.org/10.1002/jmv.20902
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Replication of Hepatitis C Virus (HCV) RNA in Mouse Embryonic Fibroblasts: Protein Kinase R (PKR)-Dependent and PKR-Independent Mechanisms for Controlling HCV RNA Replication and Mediating Interferon ActivitiesJournal of Virology, 2006
- Cell-based models of sustained, interferon-sensitive hepatitis C virus genotype 1 replicationJournal of Virological Methods, 2006
- MxA and PKR Expression in Chronic Hepatitis CJournal of Interferon & Cytokine Research, 2004
- Regulation of Hepatitis C Virus Replication by Interferon Regulatory Factor 1Journal of Virology, 2004
- Mechanism of the interferon alpha response against hepatitis C virus repliconsVirology, 2004
- Cytopathic and Noncytopathic Interferon Responses in Cells Expressing Hepatitis C Virus Subgenomic RepliconsJournal of Virology, 2003
- Hepatitis C Virus IRES-Dependent Translation Is Insensitive to an eIF2α-Independent Mechanism of Inhibition by Interferon in Hepatocyte Cell LinesVirology, 2002
- Hepatitis C virus replication is directly inhibited by IFN-α in a full-length binary expression systemProceedings of the National Academy of Sciences of the United States of America, 2001
- Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cellsNature, 2001
- The Double-Stranded RNA-Dependent Protein Kinase PKR: Structure and FunctionJournal of Interferon & Cytokine Research, 1997