Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF): EVIDENCE FOR A ROLE OF THE TNF RECEPTOR-ASSOCIATED DEATH DOMAIN PROTEIN
Open Access
- 13 July 2001
- journal article
- Published by Elsevier BV
- Vol. 276 (28), 25939-25945
- https://doi.org/10.1074/jbc.m104014200
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- More than one way to die: apoptosis, necrosis and reactive oxygen damageOncogene, 1999
- Caspases: Enemies WithinScience, 1998
- Characterization of seven murine caspase family membersFEBS Letters, 1997
- Activation of the CPP32 Apoptotic Protease by Distinct Signaling Pathways with Differential Sensitivity to Bcl-xLPublished by Elsevier BV ,1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- Molecular mechanisms of tumor necrosis factor‐induced cytotoxicityFEBS Letters, 1994
- High-dose recombinant tumor necrosis factor alpha in combination with interferon gamma and melphalan in isolation perfusion of the limbs for melanoma and sarcoma.Journal of Clinical Oncology, 1992
- Lithium chloride potentiates tumor necrosis factor-mediated cytotoxicity in vitro and in vivo.Proceedings of the National Academy of Sciences of the United States of America, 1989
- The toxic effects of tumor necrosis factor in vivo and their prevention by cyclooxygenase inhibitors.Proceedings of the National Academy of Sciences of the United States of America, 1987
- An endotoxin-induced serum factor that causes necrosis of tumors.Proceedings of the National Academy of Sciences of the United States of America, 1975