Curcumin attenuates inflammatory response in IL-1β-induced human synovial fibroblasts and collagen-induced arthritis in mouse model
- 1 May 2010
- journal article
- Published by Elsevier BV in International Immunopharmacology
- Vol. 10 (5), 605-610
- https://doi.org/10.1016/j.intimp.2010.02.011
Abstract
No abstract availableFunding Information
- Ministry of Education, Science and Technology (2009-0068626)
- National Research Foundation of Korea
This publication has 32 references indexed in Scilit:
- Inhibition of IL-1β-mediated inflammatory responses by the IκBα super-repressor in human fibroblast-like synoviocytesBiochemical and Biophysical Research Communications, 2009
- Analysis of TNFAIP3, a feedback inhibitor of nuclear factor-κB and the neighbor intergenic 6q23 region in rheumatoid arthritis susceptibilityArthritis Research & Therapy, 2009
- Potential therapeutic effects of curcumin, the anti-inflammatory agent, against neurodegenerative, cardiovascular, pulmonary, metabolic, autoimmune and neoplastic diseasesThe International Journal of Biochemistry & Cell Biology, 2008
- The roles of the classical and alternative nuclear factor-kappaB pathways: potential implications for autoimmunity and rheumatoid arthritisArthritis Research & Therapy, 2008
- TNF-Alpha and IL-1Beta-Mediated Regulation of MMP-9 and TIMP-1 in Human Glomerular Mesangial CellsNephron Experimental Nephrology, 2007
- Integrating cell-signalling pathways with NF-κB and IKK functionNature Reviews Molecular Cell Biology, 2007
- Differential induction and regulation of matrix metalloproteinases in osteoarthritic tissue and fluid synovial fibroblastsOsteoarthritis and Cartilage, 2004
- Collagen-induced arthritis development requires αβ T cells but not γδ T cells: studies with T cell-deficient (TCR mutant) miceInternational Immunology, 1999
- Structure, Regulation and Function of NF-kappaBAnnual Review of Cell Biology, 1994
- Cytokines in chronic inflammatory arthritis. V. Mutual antagonism between interferon-gamma and tumor necrosis factor-alpha on HLA-DR expression, proliferation, collagenase production, and granulocyte macrophage colony-stimulating factor production by rheumatoid arthritis synoviocytes.JCI Insight, 1990