A new model for prenatal brain damage: I. GABAA receptor activation induces cell death in developing rat hippocampus
- 30 June 2003
- journal article
- Published by Elsevier BV in Experimental Neurology
- Vol. 181 (2), 258-269
- https://doi.org/10.1016/s0014-4886(03)00053-0
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Caspase-3 Activation after Neonatal Rat Cerebral Hypoxia-IschemiaNeonatology, 2001
- Differential Involvement of L-Type Calcium Channels in Epileptogenesis of Rat Hippocampal Slices during OntogenesisNeurobiology of Disease, 2000
- Neuronal apoptosis in acute necrotic insults: why is this subject such a mess?Trends in Neurosciences, 1999
- Glutamate‐induced mitochondrial depolarisation and perturbation of calcium homeostasis in cultured rat hippocampal neuronesThe Journal of Physiology, 1999
- Vulnerability of CA1 neurons to glutamate is developmentally regulatedDevelopmental Brain Research, 1996
- Developmental Changes in Intracellular Calcium Regulation in Rat Cerebral Cortex during HypoxiaJournal of Cerebral Blood Flow & Metabolism, 1993
- Excitotoxic cell deathJournal of Neurobiology, 1992
- Comparative Effect of Transient Global Ischemia on Extracellular Levels of Glutamate, Glycine, and γ‐Aminobutyric Acid in Vulnerable and Nonvulnerable Brain Regions in the RatJournal of Neurochemistry, 1991
- Calcium Accumulation during the Evolution of Hypoxic—Ischemic Brain Damage in the Immature RatJournal of Cerebral Blood Flow & Metabolism, 1988
- Molecular Pharmacology of the Calcium ChannelJournal of Cardiovascular Pharmacology, 1984