PPARγ agonist rosiglitazone is neuroprotective after traumatic brain injury via anti-inflammatory and anti-oxidative mechanisms
- 9 December 2008
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1244, 164-172
- https://doi.org/10.1016/j.brainres.2008.09.074
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Pharmacological strategies for neuroprotection in traumatic brain injury.Mini-Reviews in Medicinal Chemistry, 2008
- JAK2 and STAT3 activation contributes to neuronal damage following transient focal cerebral ischemiaJournal of Neurochemistry, 2006
- Cytidinediphosphocholine treatment to decrease traumatic brain injury—induced hippocampal neuronal death, cortical contusion volume, and neurological dysfunction in ratsJournal of Neurosurgery, 2003
- Administration of Either Anti-Intercellular Adhesion Molecule-1 or a Nonspecific Control Antibody Improves Recovery after Traumatic Brain Injury in the RatJournal of Neurotrauma, 2002
- Protective action of the peroxisome proliferator‐activated receptor‐γ agonist pioglitazone in a mouse model of Parkinson's diseaseJournal of Neurochemistry, 2002
- Thermal Preconditioning and Heat-Shock Protein 72 Preserve Synaptic Transmission during Thermal StressJournal of Neuroscience, 2002
- Thiazolidinediones: A Comparative Review of Approved UsesDiabetes Technology & Therapeutics, 2000
- Peroxisome proliferator-activated receptors: insight into multiple cellular functionsMutation research. Reviews in mutation research, 2000
- Traumatic Brain Injury Leads to Increased Expression of Peripheral-Type Benzodiazepine Receptors, Neuronal Death, and Activation of Astrocytes and Microglia in Rat ThalamusExperimental Neurology, 2000
- Small Stress Proteins as Novel Regulators of ApoptosisPublished by Elsevier BV ,1996