Transmembrane Protein 16A (TMEM16A) Is a Ca2+-regulated Cl– Secretory Channel in Mouse Airways
Open Access
- 1 May 2009
- journal article
- Published by Elsevier BV
- Vol. 284 (22), 14875-14880
- https://doi.org/10.1074/jbc.c109.000869
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- The transmembrane protein TMEM16A is required for normal development of the murine tracheaDevelopmental Biology, 2008
- Congenital tracheal malformation in cystic fibrosis transmembrane conductance regulator‐deficient miceThe Journal of Physiology, 2008
- Calcium-dependent chloride conductance in epithelia: is there a contribution by Bestrophin?Pflügers Archiv - European Journal of Physiology, 2007
- Evidence that CFTR is expressed in rat tracheal smooth muscle cells and contributes to bronchodilationRespiratory Research, 2006
- Mechanism of the Inhibition of Ca2+-Activated Cl− Currents by Phosphorylation in Pulmonary Arterial Smooth Muscle CellsThe Journal of general physiology, 2006
- REGULATION OF NORMAL AND CYSTIC FIBROSIS AIRWAY SURFACE LIQUID VOLUME BY PHASIC SHEAR STRESSAnnual Review of Physiology, 2006
- CFTR as a cAMP-Dependent Regulator of Sodium ChannelsScience, 1995
- Anomalies in ion transport in CF mouse tracheal epitheliumAmerican Journal of Physiology-Cell Physiology, 1994
- Regulation of Cl- channels in normal and cystic fibrosis airway epithelial cells by extracellular ATP.Proceedings of the National Academy of Sciences of the United States of America, 1992
- Regulation of transepithelial ion transport and intracellular calcium by extracellular ATP in human normal and cystic fibrosis airway epitheliumBritish Journal of Pharmacology, 1991