PGC1α expression is controlled in skeletal muscles by PPARβ, whose ablation results in fiber-type switching, obesity, and type 2 diabetes
Open Access
- 1 November 2006
- journal article
- research article
- Published by Elsevier BV in Cell Metabolism
- Vol. 4 (5), 407-414
- https://doi.org/10.1016/j.cmet.2006.10.003
Abstract
No abstract availableThis publication has 33 references indexed in Scilit:
- Adiponectin and adiponectin receptors in insulin resistance, diabetes, and the metabolic syndromeJCI Insight, 2006
- Metabolic control through the PGC-1 family of transcription coactivatorsCell Metabolism, 2005
- Temporally controlled targeted somatic mutagenesis in skeletal muscles of the mousegenesis, 2005
- PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic SteatosisPLoS Biology, 2005
- How Obesity Causes Diabetes: Not a Tall TaleScience, 2005
- Cardiovascular Risk Factors Emerge After Artificial Selection for Low Aerobic CapacityScience, 2005
- Regulation of Muscle Fiber Type and Running Endurance by PPARδPLoS Biology, 2004
- Molecular basis of skeletal muscle plasticity-from gene to form and functionReviews of physiology, biochemistry and pharmacology, 2003
- Mechanisms Controlling Mitochondrial Biogenesis and Respiration through the Thermogenic Coactivator PGC-1Cell, 1999
- Detection of Retinoid X Receptors Using Specific Monoclonal and Polyclonal AntibodiesBiochemical and Biophysical Research Communications, 1994