Gene expression shift towards normal B cells, decreased proliferative capacity and distinct surface receptors characterize leukemic blasts persisting during induction therapy in childhood acute lymphoblastic leukemia
- 1 March 2007
- journal article
- research article
- Published by Springer Science and Business Media LLC in Leukemia
- Vol. 21 (5), 897-905
- https://doi.org/10.1038/sj.leu.2404613
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Expression of Late Cell Cycle Genes and an Increased Proliferative Capacity Characterize Very Early Relapse of Childhood Acute Lymphoblastic LeukemiaClinical Cancer Research, 2006
- Distinct gene expression profiles determine molecular treatment response in childhood acute lymphoblastic leukemiaBlood, 2005
- Prediction of immunophenotype, treatment response, and relapse in childhood acute lymphoblastic leukemia using DNA microarraysLeukemia, 2004
- Classification of pediatric acute lymphoblastic leukemia by gene expression profilingBlood, 2003
- Microarray Analysis Uncovers the Induction of the Proapoptotic BH3-only Protein Bim in Multiple Models of Glucocorticoid-induced ApoptosisPublished by Elsevier BV ,2003
- Analysis of gene expression patterns during glucocorticoid-induced apoptosis using oligonucleotide arraysBiochemical and Biophysical Research Communications, 2002
- Identification of genes regulated by Dexamethasone in multiple myeloma cells using oligonucleotide arraysOncogene, 2002
- Gene expression profiles of proliferating vs. G1/G0 arrested human leukemia cells suggest a mechanism for glucocorticoid‐induced apoptosisThe FASEB Journal, 2001
- Mitochondria-dependent apoptosis and cellular pH regulationCell Death & Differentiation, 2000
- Cytoreduction and prognosis in childhood acute lymphoblastic leukemia.Journal of Clinical Oncology, 1996