Cellular and molecular mechanisms of stress-induced premature senescence (SIPS) of human diploid fibroblasts and melanocytes
Top Cited Papers
- 1 October 2000
- journal article
- review article
- Published by Elsevier BV in Experimental Gerontology
- Vol. 35 (8), 927-945
- https://doi.org/10.1016/s0531-5565(00)00180-7
Abstract
No abstract availableKeywords
This publication has 89 references indexed in Scilit:
- Melanin Accumulation Accelerates Melanocyte Senescence by a Mechanism Involving p16INK4a/CDK4/pRB and E2F1Annals of the New York Academy of Sciences, 2000
- EMBO WORKSHOP REPORT: Molecular and cellular gerontology Serpiano, Switzerland, September 18-22, 1999The EMBO Journal, 2000
- Identification of a Gene That Reverses the Immortal Phenotype of a Subset of Cells and Is a Member of a Novel Family of Transcription Factor-Like GenesMolecular and Cellular Biology, 1999
- DNA Repair and Transcription in Human Premature Aging DisordersJournal of Investigative Dermatology, 1998
- Extension of Life-Span by Introduction of Telomerase into Normal Human CellsScience, 1998
- The Telomere Lengthening Mechanism in Telomerase-Negative Immortal Human Cells Does Not Involve the Telomerase RNA SubunitHuman Molecular Genetics, 1997
- Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblastsProceedings of the National Academy of Sciences of the United States of America, 1996
- Different Mechanisms of tert-Butyl Hydroperoxide-Induced Lethal Injury in Normal and Tumor ThymocytesArchives of Biochemistry and Biophysics, 1994
- tert-Butyl Hydroperoxide-Induced [Ca2+]i in Thymus and Thymoma CellsBiochemical and Biophysical Research Communications, 1993
- Oxygen modulates growth of human cells at physiologic partial pressures.The Journal of Experimental Medicine, 1984