Non–cell autonomous influence of MeCP2-deficient glia on neuronal dendritic morphology
- 22 February 2009
- journal article
- research article
- Published by Springer Science and Business Media LLC in Nature Neuroscience
- Vol. 12 (3), 311-317
- https://doi.org/10.1038/nn.2275
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- MeCP2, a Key Contributor to Neurological Disease, Activates and Represses TranscriptionScience, 2008
- Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS miceProceedings of the National Academy of Sciences of the United States of America, 2008
- Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genesProceedings of the National Academy of Sciences of the United States of America, 2007
- Glial cells as intrinsic components of non-cell-autonomous neurodegenerative diseaseNature Neuroscience, 2007
- MeCP2 Controls Excitatory Synaptic Strength by Regulating Glutamatergic Synapse NumberNeuron, 2007
- Partial rescue of MeCP2 deficiency by postnatal activation of MeCP2Proceedings of the National Academy of Sciences of the United States of America, 2007
- Metabolic Fingerprints of Altered Brain Growth, Osmoregulation and Neurotransmission in a Rett Syndrome ModelPLOS ONE, 2007
- Brain magnetic resonance study of Mecp2 deletion effects on anatomy and metabolismBiochemical and Biophysical Research Communications, 2006
- The Methyl-CpG Binding Transcriptional Repressor MeCP2 Stably Associates with Nucleosomal DNABiochemistry, 1999
- Preparation of separate astroglial and oligodendroglial cell cultures from rat cerebral tissue.The Journal of cell biology, 1980